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Gottfried Treviranus Facharzt FMH
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PERIPHERAL CAUSES OF BIPOLARITY (...WPA Athens... IRBD...)

This projects starts from the assumption than intrinsic primary causes of severe brain dysfunction are unlikely, and that the "real biology" of peripheral processes is unduly neglected.
PERIPHERAL CAUSES OF BIPOLARITY: WPA Athens 2012 to 2013
3 posters at the The Multidisciplinary Facets of Psychiatry WPA conference at Athens Watch them on www.prezi.com!
IRBD 2012 presentation on the pterygo-palatine ganglion
How the pterygo-palatine ganglion could matter to bipolar disorder too. By the end of the week you will find a referenced version of the talk with active OpenAccess links. thereafter visitors of IRBD2012 will be offered direct access to so-called living documents, which allow elaboration of further steps.
IRBD 2011 Brain architecture & K. Askland's biaxial model
This poster was shown at the IRBD 2011 conference in Rome. It deals with predictions derived by crossing the biaxial model of K. Askland for bipolar disorder with the neurobiological anatomic interpretation of the THOUGHT-ACTION-MOOD-model. A general stronger dependance of frontal symbolic and mesocephalic incentive appetite ("THOUGHT") from receptors and transmitters is opposed by a stronger dependance of the "parietal movement brain" (ACTION/INTUITION) from ion channels - and their relative genes.
IRBD 2013 presentation on mast cells & ... Bartonella.
This presentation is a first attempt to sketch a unified framework how a set of clinical psychiatric observations and recent advances in biomedicine can provide possible answers to the etiology e.g. of bipolar disorders and of comorbid inflammatory conditions. Mast cells (MCs) are the first line in defense against germs of which e.g. Bartonella is known to change the genome ... eventually also of MCs. MCs can be genetically changed to cause Mastcell Activation Disorders, which show "mixed" pathologies.MCs advance through the lymphatics which are permeabilized to hasten antigen transport. MCs and their pellets then follow non-canonical conduits to the brain. MCs have 100 different receptors and their behavior are lstingly primed to provide highly speciific hubs at predetermiend adresses.
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